![]() What is the difference between the primary auditory cortex and auditory association area? What happens if your Broca’s area is damaged?.What neurological processes are taking place when a person is hearing music?.What symptom would present if a person damaged the prefrontal cortex?.What is auditory attention in psychology?.What is the auditory cortex responsible for?. ![]() How do we process auditory information?.What are the three regions of the auditory system?.What is the function of the Wernicke area?.What part of the brain controls auditory?.What would happen if the auditory cortex was damaged?.What is the function of the auditory association area?.Where is your auditory association area?.What is the difference between the primary auditory cortex and auditory association area?.This study can guide further research on design of therapeutic strategies to encourage adaptive changes and discourage maladaptive changes after brain damage, sensory/motor dysfunction, and deafferentation. These findings provide insight into compensatory plasticity after sensory dysfunction and brain damage and new information about the role of inhibition in cross-modal plasticity. The reorganization of brain areas after recovery from brain damage may require stronger inhibition in order to process multiple sensory modalities simultaneously. These results suggest that compensatory plasticity can be maladaptive and thus impair the recovery of the original sensory cortical function. I found an increase rather than a decrease of inhibition in the rewired auditory cortex, suggesting that broader tuning curves in the experimental animals are not caused by loss of lateral inhibition. Because lateral inhibition is proposed to contribute to refinement of sensory maps and tuning of receptive fields, I tested whether loss of inhibition is responsible for the compromised auditory function in my experimental animals. Auditory tuning curves were broader and tonotopic maps were disrupted in the experimental animals. As might be predicted from the fact that auditory neurons and visual neurons were intermingled throughout the entire auditory cortex, I found that residual auditory tuning and tonotopy in the rewired auditory cortex were compromised. The distribution of these different neuronal responses did not show any clustering or segregation. I found that auditory cortex contained auditory, visual, and multisensory neurons after it recovered from neonatal midbrain damage (Mao et al. I addressed maladaptive changes of auditory cortex after invasion by ectopic visual inputs. In this thesis, I studied the function of auditory cortex after recovery from neonatal midbrain damage as a model system that resembles patients with brain damage or sensory dysfunction. We are interested in general mechanisms that underlie compensatory plasticity after brain damage, particularly when multiple brain areas or multiple modalities are included. The success of recovery from brain injury involves functional compensation in the affected brain area. Brain injury is a serious clinical problem.
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